PT106.S3.Q25 > Question

SECTION 1 · lr

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SECTION 2 · lr

SECTION 3 · lr

SECTION 4 · rc

PASSAGE 1

Question 1 Question 2 Question 3 Question 4 Question 5

PASSAGE 2

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PASSAGE 3

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PASSAGE 4

Question 22 Question 23 Question 24 Question 25 Question 26

The author hypothesizes that the size of the interstitial nucleas determine whether a male cat can contract disease X. This is based on data showing that male cats who died from disease X have larger interstitial nuclei than those male cats that didn’t die from disease X.

The author assumes that the correlation observed between the size of the interstitial nucleus in male cats and death from disease X must be explained by the size of the interstitial nucleus having a causal impact on the presence of disease X.

The conclusion is just about male cats’ susceptibility to disease X based on their interstitial nucleus size. Whether female cats can get disease X doesn’t impact the author’s reasoning, which was limited only to what we observed in male cats.

This establishes that many cats that get X also get Z. But this doesn’t provide any insight into the cause of disease X in male cats.

This is additional data suggesting a correlation between a larger interstitial nucleus and contracting disease X. This is consistent with the author’s reasoning.

This shows that a tiny percent of male cats without disease X had a larger interstitial nucleus. But we already know that all the male cats with disease X had this larger feature. So, (D) shows that there is a correlation between disease X and a larger interstitial nucleus.

The first sentence told us that the interstitial nucleus is a part of the hypothalamus. If the hypothalamus isn’t known to be a cause of Y, which includes subtype disease X, this suggests the interstitial nucleus isn’t a cause of disease X.

PrepTest 106 - Section 3 - Question 25

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