PT117.S2.Q20 > Question

SECTION 1 · rc

PASSAGE 1

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PASSAGE 3

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PASSAGE 4

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SECTION 2 · lr

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SECTION 3 · lr

SECTION 4 · lr

The author concludes that a certain gene variant causes an inclination toward thrill-seeking behavior. This is based on the fact that the author’s research indicates that children who engage in impulsive behavior similar to adult thrill-seeking behavior are twice as likely as other children to have that gene variant.

The author assumes that a correlation between the gene variant and impulsive behavior proves a causal relationship. The author also assumes that the cause of impulsive behavior in children also causes adult thrill-seeking behavior.

The author never suggested every adult has the gene variant. So, the fact many impulsive adults aren’t unusually sensitive (and therefore might not have the gene variant) doesn’t undermine the author’s reasoning.

This shows that the alleged correlation shown by the author’s research doesn’t reliably tell us about impulsive behavior. If we can’t be sure that the author’s research identified impulsive behavior, that reduces the support provided by the research for a causal relationship.

The argument is about alleged impulsive behavior in children and adult thrill-seeking behavior. Whether children’s behavior is called thrill-seeking doesn’t affect the potential cause of impulsive behavior or adult thrill-seeking behavior.

The author never suggested that every child with impulsive behavior grows up to exhibit adult thrill-seeking behavior. So, even if many adults end up not impulsive and not thrill-seeking, that doesn’t undermine the underlying correlation the author observed.

This suggests the gene variant might cause other types of behavior in addition to thrill-seeking behavior. But it doesn’t suggest the gene variant might not cause thrill-seeking behavior. (E) could have weakened if the thrill-seeking was correlated with a different gene.

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